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103 Furthermore, blocking neutrophil recruitment is also a feasible treatment approach

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Human multi-omics evidence suggests persistent mitochondrial suppression after SARS-CoV-2 infection Human COVID-19 autopsy data support region-specific neuroimmune and bioenergetic disruption Building on our previous observations of persistent OXPHOS suppression across multiple COVID-19 autopsy tissues ( Figure 1A ), we next examined whether similar mitochondrial and immune transcriptional programs are evident in the CNS

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