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20 Possible mechanisms are: natriuresis, due to increased atrial natriuretic peptide secretion, GLP-1R activation in the proximal nephron tubule and Na + /H + transporter inhibition (which reduces sodium reabsorption), as well as vasodilation due to nitric oxide-dependent vasodilation (endothelial GLP-1R activation) and direct effects on smooth muscle cells GLP-1R

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However, due to the essential nature of iron for many biological processes, chelation therapy is typically reserved for preventing iron overload in patients requiring frequent blood transfusions, such as those with sickle cell disease and thalassemia [229]

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Treatment options for gastritis also vary depending on the cause