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Examples include the upregulation of cyclic adenosine monophosphate-responsive element binding protein-regulated transcription coactivator (CRTC) 2 and phosphorylated acetyl-CoA carboxylase (p-ACC), or downregulation of peroxisome proliferator-activated receptor (PPAR) , acetyl-CoA carboxylase (ACC), and sterol regulatory element binding protein 1 (SREBP1) (63)

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Additionally, studies in patients with T2DM are now needed to explore potential clinical benefits [197]