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[168], he proposed that NO may bind to the enzymes of mitochondrial ETC, such as NADH succinate oxidoreductase, NADH ubiquinone oxidoreductase, and cis aconitase and induce uncoupling of OXPHOS followed by the inhibition of glycolysis [66] and depletion of ATP in the cells [169]

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The dual nature of NR4a1s activity necessitates a balanced approach to its modulation, reflecting the variable trends observed across different experimental models

Liu, H., Dear, A