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[56] Senescent cells secrete a harmful mix of inflammatory molecules known as the senescence-associated secretory phenotype (SASP)

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Despite considerable progress in our understanding of MASH, the particular mechanisms underlying disease development and the variable progression among patients with similar clinical risk factors remain inadequately explained

Daungkamon Nokinsee et al

1145 However, their dysfunction is a catalyst for cardiovascular disease progression, as evidenced by significant mitochondrial disruptions during the early stages of these diseases