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(2023) utilized an integrated network pharmacological approach alongside RNA-Seq methods to investigate transcriptional alterations triggered by PF in Cis-induced AKI, revealing that PF reduces cellular death and inflammatory responses in Cis-induced AKI via enhancement of Hsp90AA1-Akt protein-protein interactions

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Dysregulated mitochondrial metabolism promotes T cell exhaustion Antigen stimulation is associated with impaired mitochondrial OXPHOS function Under prolonged antigenic stimulation, T cells exhibit impaired mitochondrial OXPHOS function, leading to increased reliance on glycolytic processes for ATP production [24]