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Our combination therapy of (-)-Vestitol and Salviolone, identified via network medicine, targets multiple AD-related pathways, including Neuroactive ligand-receptor interaction and Calcium signaling pathway, potentially offering a more comprehensive, disease-modifying approach

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Experimental studies involving diabetic ulcer models have suggested accelerated wound closure and improved tissue regeneration following GHK-Cu administration, supporting continued investigation into connective tissue repair and extracellular matrix restoration