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In another study, it was found that after 6 weeks of swimming exercise, the level of HSP70 in the hippocampus and prefrontal cortex was upregulated and the behavioral changes and the function of the HPA axis were improved in chronic unpredicted mild stress (CUMS) animals, while the level of inducible nitric oxide synthase (iNOS) was downregulated [70], suggesting that previous exercise can delay the disease progression characterized by TBI-induced movement defects and memory impairment through the activation of endogenous antioxidant molecules such as Nrf2 and HSP70 to protect the targets of oxidative stress (such as Na + - K + - ATPase) after TBI

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The discovery of erythronecroptosis should, therefore, drive research on immunogenicity of erythrocyte cell death