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2017 Jan 15;440:106-115 16.1 Duntas LH

In: Physiologic and pathologic angiogenesis-signaling mechanisms and targeted therapy

Excessive burden of ROS, failure of clearance mechanisms and even lack of antioxidants may lead to the accumulation of ROS and to oxidative stress [40], which are associated with genetic damage and breast cancer onset [61,62,63,64,65]

In order to substantiate the therapeutic potential of JMF4073 for TKI-resistant T315I-Bcr-Abl cells, we investigated the mechanism responsible for the differential response to JMF4073

The irritancy threshold in animal testing was also above 30% (so real-world formulas sit far below anything concerning)