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GLP-1 analogs have been shown to modulate neurotransmitter release related to hunger and reward in the hypothalamus and striatum, resulting in reduced food intake [13]

Semaglutide activates GLP-1 receptors throughout the body at a sustained, pharmacological level, producing a range of metabolic effects that collectively make meaningful, sustained weight loss biologically achievable

Elevated ROS increases PRDX2 monomer formation, which causes TIMELESS-TIPIN dissociation from the replisome and replication slowdown, thereby inducing replication stress