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10.1016/j.jocn.2020.09.054 J

TB-500 may therefore be hypothesized to encourage angiogenesis by pairing a cytoskeleton-linked rise in endothelial motility with signaling shifts that elevate pro-angiogenic programs such as VEGF-A and Ang2/Tie2 through Notch/NF-B coupling in damaged cellular structures

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We have reported that SodA and TpxD are regulated by the StkP-ComE signal transduction pathway and that are essential for intracellular survival of the pneumococcus within pneumocytes [7]