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Additionally, the excessive production of ROS creates a vicious cycle that further exacerbates inflammation and oxidative stress, contributing to the syndrome [10, 11]

The primary driver of weight loss appears to be reduced caloric intake secondary to appetite suppression and delayed gastric emptying, rather than a substantial increase in basal metabolic rate

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Therefore, caution is advised when prescribing these agents to individuals with pre-existing gallbladder conditions, despite the lack of Japan-specific evidence [13]