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pharmacological targeting of which substantially delays cardiomyocyte maturation in postnatal hearts, markedly enhances cardiomyocyte proliferation, and improves cardiac function in infarcted hearts

This results in increased airway resistance and responsiveness, potentially due to changes in airway smooth muscle (ASM) contractility

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In contrast, 1907 demonstrated two binding sites ( K D1 = 0.8 0.6 M, K D2 = 8.7 3.3 M), with a 100-fold increased affinity over LD2, and a tenfold improved K i of 5.8 4.6 M by FP (Table 1 and Fig