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We next investigated the mechanism underlying autoproteolytic processing and whether modulation of SnCE1 autoproteolytic processing can be attributed to individual lysine acetylation sites, as demonstrated for SnCE1 oligomerization

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The metabolic impact on histone acetylation and transcription in ageing

Jelovac[23] found that BPC 157 acted in favor of the natural homeostasis of the GABA receptor complex and of the GABAergic transmission, thus having a mechanism at least partly different from those involving diazepam tolerance/withdrawal