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Comparative evaluation of IGF-I gene transfer and IGF-I protein administration for enhancing skeletal muscle regeneration after injury

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These therapies target neurotransmitter systems, neuroplasticity, and mitochondrial function within the brain

Other mechanisms In addition to the aforementioned well-established therapies, numerous other pharmacological agents, chemical compounds, proteins, and methodologies that have not yet been implemented in clinical practice are currently being investigated to restore the Th17/Treg balance to treat AR (Table 6)

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