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set out to determine whether enteroendocrine cells (EECs) could sense circulating LPS and thus release GLP-1 in order to modulate the pro-inflammatory response6

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They investigated the relationship between intestinal flora imbalance and severe MASLD lesions (MASH and fibrosis) and showed that an increase in the number of Ruminococcus was positively associated with an aggravation of the degree of fibrosis (76)

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