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Membrane-incorporated PUFAs serve as primary targets for ROS-mediated oxidation, with their oxidative susceptibility increasing proportionally to the number of conjugated double bonds (Rouzer and Marnett, 2003), following the initiation of oxidation, free radicals exhibit dual propagation mechanisms: intramolecular migration within lipid bilayers or intermolecular chain reactions amplifying oxidative damage (Samovich et al., 2024)

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Thus, treatment strategies for patients with NAFLD with concomitant T2DM are important to prevent the progression of NAFLD

A2: In vitro studies suggest that Epithalon can activate telomerase in certain cell types and support telomere maintenance, making it a useful tool compound for exploring how telomere biology relates to genomic stability and cellular aging mechanisms

prophylaxis of DVT which may lead to PE in patients undergoing knee or hip replacement surgery