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Administrated of N -acetylcysteine (NAC), a chemically synthesized prodrug of Cys, is used to increase intracellular GSH levels, because NAC is easily transported into cells and successively converted to Cys and GSH through -EC [10]

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The buffering of antioxidant species can be accomplished by reduced glutathione (GSH), that is also required for phase II detoxification in which endogenous and exogenous toxic metabolites are conjugated to GSH for their removal [10], [11]