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The chronic activation of the type I IFN pathway by oxidative stress contributes to the dysregulation of immune responses and the production of autoantibodies in SLE [146]

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Once activated, the mentioned pathways encourage growth hormone release, which in turn modulates downstream signals, insulin-like growth factor 1 (IGF-1), and other metabolic factors

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These copper ions are crucial for enzymatic processes involved in tissue repair, collagen synthesis, and antioxidant defense