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We note that because Cr GPDH does not require O 2 as a co-substrate our new genetic tools can be used in various applications in hypoxia as it allows to decrease cellular NADH/NAD + under these conditions ( Figure 4i )

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Many users find that metabolic benefits persist for several weeks after discontinuing treatment, suggesting lasting improvements in cellular function

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We reasoned that as the GluC cleavage specificity is different from the three catalytic activities of the proteasome, considering that the proteasome caspase-like site cleaves more efficiently after aspartic acid residues than glutamic acid (Kisselev et al, 2003), we could distinguish proteasome-generated peptides from those produced by GluC