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Evidence and risk depend on how it is used

The skin-specific mechanisms of GHK-Cu include stimulation of Type I and Type III procollagen synthesis, increased production of dermatan sulfate and other glycosaminoglycans, activation of the enzyme lysyl oxidase (which cross-links newly formed collagen), reduction of matrix metalloproteinase-1 (MMP-1, collagenase) activity, and anti-inflammatory suppression of TNF-alpha-induced IL-6 secretion

The optimal ghk-cu protocol depends entirely on the therapeutic goal, distinguishing between systemic effects and localized treatment

A rebound in glutamate activity, increasing brain excitability 16

By providing your skin niacinamide, you're recharging your skin's NAD levels