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Administration of spironolactone, a selective MR antagonist, counteracted stress-induced mGlu2 suppression and the related development of depressive-like behavior, implicating rapid action of glucocorticoids via a receptor that is known to modulate glutamate release [20]

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It works by: Precise Action: Slowly releasing hydroquinone molecules at the site of melanocytes, directly inhibiting tyrosinase with minimal irritation or risk of surrounding-skin lightening (ochronosis)