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The dual growth hormone releasing peptide mechanism is one reason clinicians favor this combination over older peptide protocols

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Exogenous androgen protects ETC function by alleviating mitochondrial oxidative stress, as evidenced by results showing that testosterone supplementation reduces Nox1 expression, and increases the expression levels of antioxidants (e.g., SOD2, GPX1, thioredoxin, catalase, and glutathione reductase) in the ventral prostate of castrated rats (84, 126)

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