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Subsequent increases in the permeability of the intestinal barrier in CKD patients promote the translocation of bacterial products of intestinal origin, such as LPS, uremic toxins, and cytokines, into the systemic circulation, resulting in local and systemic inflammation, as well as oxidative stress associated with CKD (Krukowski et al., 2023

Mechanistic reviews by Chelakkot et al., published in Experimental & Molecular Medicine (2018) , describe how dysregulation of these junctions is associated with increased intestinal permeability and immune activation

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