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These results indicate that neuroinflammation is strongly associated with neuronal death in GCLC-KO mice, particularly in the early stages of brain atrophy

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In parallel, these peripheral inflammatory signals and microbial metabolites can breach the bloodbrain barrier (BBB) or activate afferent vagal pathways, leading to neuroinflammation in metabolic brain centers such as the hypothalamus and nucleus tractus solitarius (NTS) (224)

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