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Research of novel drug treatments for diabetes has thus moved away from insulin analogs to new peptide hormones that have similar effects, in particular the incretin hormones, such as Glucagon-like peptide-1 (GLP-1), glucose-dependent insulinotropic polypeptide (GIP) and Dipeptidyl peptidase-4 (DPP-4), a key enzyme responsible for cleaving and inactivating both of GLP-1 and GIP peptides at the penultimate alanine residue ( GLP-1 is part of the peptide growth-factor family and activates a glucagon-type seven membrane-spanning G-protein coupled receptor

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The conclusive proof: the effect is abolished in animals CD36 knockout closest preserved in animals GHS-R1a knockout This means that the cardioprotective effect is mediated exclusively by CD36, not by the classical GH axis

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