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The resistance to lipase also prevents hydrolysis catalyzed by bacterial lipases in the colon so that irritant fatty acids are not produced

This age-related decline was first characterized through copper-binding peptide fractionation studies of human serum and correlates temporally with decreased wound healing capacity, reduced collagen synthesis rates, and diminished antioxidant enzyme activity observed in aging tissues

Notably, this heat stroke-induced LIRI differs fundamentally from reperfusion injury in hypovolemic shock or surgical contexts: in heat stroke, reperfusion is preceded by hyperthermia-induced endothelial damage and systemic inflammatory priming, which amplify oxidative stress (e.g., xanthine oxidase activation) and neutrophil infiltration