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Metabolic reprogramming mainly occurs in the mitochondria and cytoplasm, which refers to the metabolic changes made by cells in response to various stimulus pressures

The mechanism involves mitochondrial electron transport support and antioxidant effects, though human clinical trial data remains limited compared to animal studies

Silencing of glutamate-cysteine ligase (GCLC), a rate-limiting enzyme in GSH biosynthesis, caused an increase in oxidative stress/protein oxidation and downregulation of the the CYP2R1 , CYP27A1 , CYP27B1 , VDBP , and VDR genes in cultured hepatocytes