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The expression of LPL is regulated by insulin and in its absence a hypertriglyceridemia results

Harusato A, Abo H, Ngo VL, Yi SW, Mitsutake K, Osuka S, et al

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A possible genotype-phenotype correlation has been proposed with homozygous carriers of frameshift variants affecting the MPTb domain displaying a more severe clinical course than that observed in individuals harboring single amino acid changes in the FADS domain that are potentially more responsive to riboflavin therapy [34]

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