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Recently, the purpurin analog R162 (an inhibitor of GLUD1) also showed promising results with respect to attenuating the proliferation of breast, NSCLC, and glioma cells in vitro and in patient-derived xenograft mouse models 23

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The clinical features of GLP-1 RA-associated BP may not always correspond to the characteristic tense blisters of classic BP, and elevated autoantibody levels can persist even as clinical symptoms improve

Targeting mitochondrial oxidative metabolism in melanoma causes metabolic compensation through glucose and glutamine utilization