HGF/c-Met signaling is a well-established oncogenic pathway: c-Met is amplified or overexpressed in multiple cancers (gastric, lung, renal, hepatocellular) HGF/c-Met activation promotes tumor cell proliferation, survival, invasion, and metastasis c-Met inhibitors are FDA-approved cancer drugs (cabozantinib, crizotinib, capmatinib) A compound that potentiates HGF/c-Met signaling raises theoretical concerns about promoting tumor growth or transformation with chronic use
Clinical fit: Whether peptide therapy fits the patients goals, medical history, medications, and risk profile
Those who participate in a vegan or vegetarian diet most commonly suffer from a B-12 deficiency due to it only being found in animal-based foods
Who May Benefit From Glutathione IV Therapy
The Role of the Master Antioxidant Glutathione does not just work alone