Most importantly, the procognitive/antidementia capacity of orally delivered dihexa was blocked by an intracerebroventricularly delivered HGF antagonist, as measured using the Morris water maze task of spatial learning
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Another factor is the storage conditions
The study had no placebo group, no randomisation, no blinding, no imaging-confirmed repair endpoint and no clear analytical confirmation that the second peptide was the defined TB-500 fragment
The peptide downregulates TNF- (6.2-fold reduction) and IL-6 (4.1-fold reduction) while upregulating anti-inflammatory IL-10 (8.1-fold increase)[12]
