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The isothiocyanate sulforaphane induces respiratory burst oxidase homologue D-dependent reactive oxygen species production and regulates expression of stress response genes

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60,61,62 In summary, our study demonstrated that interference with RAS signaling via combined SHP2/MEK inhibition provoked mitochondrial remodeling and oxidative stress both in vitro and in vivo, largely irrespective of the molecular PDAC subtype