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Thanks to the activation of the TLR4 pathway, Ikb- is phosphorylated and degraded by proteasome, leading to NFkB release and translocation into the nucleus to increase the expression of targeting genes implicated in the inflammatory response [36,37]

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The drug can impair the production of prostaglandins, which are essential for maintaining adequate blood flow and filtration in the kidneys

Although few meta-analyses have investigated the efficacy of GLP-1 RAs on the histologic resolution of MASLD, all have shown significant improvements

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