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Overall, these findings lead to the conclusion that GliT not only catalyzes formation of gliotoxin, but simultaneously prevents the dysregulation SAM/SAH and consequential deleterious impacts of elevated SAH, production of toxin Hcy and depletion of Met (Owens et al., 2015)

Regulation of iodothyronine deiodinases in the pax8 / mouse model of congenital hypothyroidism