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Moreover, more recently a family of proteins called peroxiredoxins has been recognized as catalyzing the reduction of H 2 O 2 by GSH and/or other thiols, but with cysteine in its thiolate form, in their active sites rather than selenium-cysteine (Dickinson and Forman, 2002)

Many mild cases improve with rest, stretching, supportive footwear, and activity changes over several months

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This imbalance can trigger the production of an array of ROS, such as NO, O 2 , H 2 O 2 , etc., which play important roles in the pathophysiological processes that drive gout inflammation [6]

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