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Collectively, the interplay between GSH, GPX4, and cellular resilience highlights the potential of GSH-targeted strategies to improve the efficacy of standard treatments and possibly alleviate drug resistance in cancer therapy

However, blocking specific enzymes related to DNL may, in some cases, exacerbate NASH and the accompanying metabolic deterioration by promoting accumulation of cytotoxic lipid species, indicating the importance of the composition of the fatty acid pool in the liver (Fig