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Interestingly, upon RNA virus (Sendai virus) and DNA virus (HSV-1) infection, PRMT1 forms oligomers to recruit and methylate the downstream effector TBK1 at R54, R134, and R228, promoting its aggregation and trans-autophosphorylation and the subsequent IRF3-mediated IFN- production [209]

Prior studies have shown that SLC7A11 inhibition, which limits intracellular cysteine availability and reduces glutathione synthesis, increases ROS accumulation, leading to tumor cell vulnerability

Heijman J, Algalarrondo V, Voigt N, Melka J, Wehrens XH, Dobrev D, Nattel S (2016) The value of basic research insights into atrial fibrillation mechanisms as a guide to therapeutic innovation: a critical analysis

And honestly, we need to talk about its packaging