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Evidence type: Human RCT | Observational | Animal | In vitro | Regulatory Mechanism of Action As a copper chelator, GHK-Cu modulates over 4,000 human genes, upregulating those involved in tissue remodeling (e.g., collagen I, III) and downregulating pro-inflammatory pathways like NF-B

I kept coming back to #2 as I listened to the meeting but someone should have raised point #1

Brings me to the verge of crying

The acid tolerance levels described are generalizations

On the other hand, the overexpression of G6PD in vascular endothelial cells decreases the accumulation of ROSs in response to exogenous and endogenous stressors, improving bioavailable NO levels [54, 55]