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Journal of Clinical Endocrinology & Metabolism (JCEM) (2006) 2
Role of G6PD-deficiency-mediated RBC hemolysis in ASCVD Any discussion of the potential impact of G6PD-d on ASCVD would be incomplete without considering how the predominant hemolytic phenotype may contribute to worsened disease
(a5) synthetic sweetening substances, preferably selected from the group consisting of betaine, sodium cyclamate or other physiologically acceptable cyclamate, acesulfame potassium or other physiologically acceptable salts, neohesperidin dihydrochalcone, naringin dihydrochalcone, saccharin sodium salt, aspartame, superaaspartame, neotame, alitame, edmuntin (advatame), perilla, sucralose, N- (4-cyanophenyl) -N- (2, 3-methylenedioxybenzyl) guanidineacetic acid, N- [ [ (3, 5-dichlorophenyl) amino ] [ (diphenylmethyl) amino ] methylene ] glycine, saccharonic acid and sulotene (sucrooctate)
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