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IGF-1 LR3 vs IGF-1 DES (1-3) vs HGH (Somatropin) vs Mecasermin (Native rhIGF-1) Feature Type IGF-1 LR3 Synthetic IGF-1 analog IGF-1 DES (1-3) Truncated IGF-1 variant HGH (Somatropin) Recombinant growth hormone Mecasermin (Increlex) Recombinant native human IGF-1 Feature Amino acids IGF-1 LR3 83 IGF-1 DES (1-3) 67 HGH (Somatropin) 191 Mecasermin (Increlex) 70 (identical to endogenous IGF-1) Feature Half-life IGF-1 LR3 20-30 hours IGF-1 DES (1-3) ~20-30 minutes HGH (Somatropin) 2-3 hours Mecasermin (Increlex) ~5.8 hours Feature Receptor potency IGF-1 LR3 ~3x native IGF-1 IGF-1 DES (1-3) ~10x native IGF-1 (locally) HGH (Somatropin) Indirect (drives endogenous IGF-1) Mecasermin (Increlex) 1x (it is native IGF-1) Feature Action IGF-1 LR3 Systemic, long-acting IGF-1 DES (1-3) Localized, rapid HGH (Somatropin) Systemic upstream cascade Mecasermin (Increlex) Systemic, replacement therapy Feature Dosing frequency IGF-1 LR3 Once daily IGF-1 DES (1-3) 2-3x daily HGH (Somatropin) 1-2x daily Mecasermin (Increlex) Twice daily SubQ Feature Common dose context IGF-1 LR3 20-100 mcg/day (research) IGF-1 DES (1-3) 50-150 mcg/day split (research) HGH (Somatropin) 2-8 IU/day (research) Mecasermin (Increlex) 0.04-0.12 mg/kg twice daily (clinical) Feature IGFBP binding IGF-1 LR3 Very low IGF-1 DES (1-3) Very low HGH (Somatropin) N/A (acts upstream) Mecasermin (Increlex) Normal (full IGFBP affinity) Feature FDA status IGF-1 LR3 Not approved IGF-1 DES (1-3) Not approved HGH (Somatropin) FDA-approved (multiple indications) Mecasermin (Increlex) FDA-approved (pediatric severe primary IGFD) Feature WADA status IGF-1 LR3 Prohibited IGF-1 DES (1-3) Prohibited HGH (Somatropin) Prohibited Mecasermin (Increlex) Prohibited Feature Practical note IGF-1 LR3 Long-acting IGF-1 analog with the most accumulated community data IGF-1 DES (1-3) Localized, rapid signaling

(2023) 18:e0280872

"That actually goes against the Hippocratic oath, which is do no harm and if we don't know if we're causing harm, that's an issue." This article was originally published on NBCNews.com

How is therapy administered

Overall, NAC can facilitate alveolar bone regeneration by promoting bone formation and inhibiting bone resorption, thus enabling a positive therapeutic effect for periodontal tissue defects caused by periodontitis