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These findings might be partly explained by short follow-up, different doses of l-carnitine, and the inclusion of low-quality trials without randomization and blinding [14, 15]

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High levels of homocysteine, with the slowing of GPx1 blood vessel activity, promote a higher concentration of intracellular peroxides that enhances oxidative stress and causes damage to endothelial cells in the pathogenesis of atherogenesis

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