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This condition, known as ischemiareperfusion (I/R) injury, is distinguished by the formation of reactive oxygen species (ROS), the activation of inflammatory mediators, and extensive tissue inflammation (Soares et al., 2019)
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The findings indicate that cerebral I/R injury not only causes a specific type of programmed neuronal death called PANoptosis but also triggers microglia to adopt a pro-inflammatory (M1) phenotype, both in living organisms and in vitro
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