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Figure 2C indicates genes with potential for substantial negative roles in CR DM mucosa
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the treated population, including patients with HCV genotype 3, compensated cirrhosis, and prior treatment experience (including treatment with SOF-based regimens), was the largest such patient group to date.[10, 11, 14] Among patients with GT3 infection and compensated cirrhosis, treatment with 12 and 16 weeks of RBV-free G/P yielded high rates of SVR12 in treatment-naive and treatment-experienced patients, respectively
