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Semaglutide activates GLP-1 receptors in your stomach, slowing the rate at which food moves into your small intestine

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Moreover, it negatively regulates the accessibility and infiltration of immune cells by affecting the metabolite availability for immune effector cells and by inducing an immunosuppressive phenotype via an upregulation of immune checkpoints on cancer cells, e.g., PD-L1 [11, 12]