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Its toxic effects extend to male reproductive health, where it adversely effects on testicular function, impairs spermatogenesis, and induces oxidative stress through mechanisms such as elevating the Reactive oxygen species concentration, mitochondrial dysfunction, and cellular apoptosis which severely affects the reproductive health (74, 75)

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R.OrlandoP.SilvestriS.CirilliI.MarcheggianiF.CiavattiniA.et al (2018)

TMEM16A in cystic fibrosis: activating or inhibiting

Unfortunately, due to the cationic nature of QuicGSH, it primarily accumulates in mitochondria