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As a hallmark of UC is neutrophilic infiltration and release of pre-formed MPO-containing granules, we examined MPO protein levels by immunohistochemistry and found significantly higher MPO levels in UC patient colons than control colons (Figure 1c)

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While GHK has been shown to inhibit the inflammatory response and epithelial-mesenchymal transition (EMT) via the TGF-1/Smad 2/3 and IGF-1 pathways in bleomycin-induced fibrosis models [24], its broader anti-inflammatory effects in the context of pulmonary fibrosis pathogenesis may warrant further exploration

doi: 10.1111/acel.12685 71 PangB.ZhaoL

(2020) 17:e1003198