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While CMV infection of myeloid cells allows for their immediate manipulation (Baasch et al, 2021), non-permissive lymphocytes like T cells and NK cells are indirectly influenced leading to the accumulation of CMV-specific CD8 + T memory cells and the induction and expansion of adaptive memory-like NK cells with distinct epigenetic and functional features (Gum et al, 2004

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Mao found that treating cancer cells with GPX4 inhibitors resulted in acute depletion of N-carbamyl-L-aspartate (C-ASP, a pyrimidine biosynthesis intermediate), accompanied by accumulation of uridine, which could be inhibited by fer-1, a ferroptosis inhibitor [18]

J Clin Dermatol