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More relevant for targeting ECD was the study of bloodendothelium interactions, where HUVECs were divided into untreated (quiescent endothelium) and stimulated with phorbol-12-myristate-13-acetate (PMA) to mimic an inflammatory response before whole blood perfusion

Meanwhile, dual agonists, which target and activate both the GLP-1 (Glucagon-like peptide-1) receptor and the GIP (glucose-dependent insulinotropic polypeptide) receptor, and triple agonists (targeting the GLP-1+GIP+Glucagon receptors) are also showing promise

To this end, we compared regions with and without lesions, referred to as respectively MS versus MS control samples

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This observation highlights that endogenous GIP is essential even in type 2 diabetes, suggesting promise for pharmacological agents that target the GIPR