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3.4 GSAAT interacts with TRX isoforms and with NTRC We used different methodological approaches to confirm the interactions of GSAAT with TRXs and NTRC

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have shown that compared with intravenous glucose, the increase of plasma insulin level after oral glucose was greater, and the high level of insulin was maintained for a longer time, which provided a basis for intestinal factors to stimulate insulin secretion (Elrick et al., 1964)

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In a comprehensive clinical overview, researchers explain that this buffering system prevents the toxic accumulation of unoxidized, short-chain acyl groups within the mitochondria